Background:
Anakinra is a recombinant form of human interleukin‑1 receptor antagonist (IL‑1Ra). Limited evidence supports use during pregnancy and breastfeeding.1 Rheumatology guidelines support use in pregnancy for disease control.2 Anakinra is not a monoclonal antibody, hence is not susceptible to Fc-receptor-mediated transplacental transfer.2 Endogenous IL‑1Ra is present in breast milk;3 however, data on placental transfer, breast milk concentrations, and infant exposure to anakinra remain limited.
Case:
A 27‑year‑old woman with Familial Mediterranean Fever achieved disease control with anakinra, which was continued during pregnancy due to high flare risk. She delivered at 39+3 weeks and breastfed. IL‑1Ra levels were measured in maternal serum during pregnancy, cord blood at delivery, breast milk, and infant serum postpartum.
Results:
The assay cannot differentiate anakinra from endogenous IL‑1Ra. At 36 weeks, maternal trough and peak IL-1Ra levels were 49,700 ng/L and 194,240 ng/L respectively, consistent with non‑pregnant populations.4 Cord blood concentration was 1,739 ng/L, within expected ranges.5 Breast milk concentrations reflected anakinra pharmacokinetics,4 with trough levels of 498 ng/L and peak levels of 8,066 ng/L at two hours post‑dose, followed by decline. Infant serum IL‑1Ra 70 days postpartum was higher than expected (7,173 ng/L), as levels are expected to rapidly decline postpartum in a well infant.6
Conclusion:
This case suggests minimal transplacental transfer of anakinra and confirms transfer into breast milk. Elevated infant IL‑1Ra may reflect oral bioavailability, although biologically implausible, or simply increased endogenous production. Further studies are needed to confirm these findings.
* These authors are co-senior authors.